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The management of osteochondral defects of the talus remains a clinical challenge
as a result of the poor intrinsic healing potential of cartilage. Osteochondral lesions
of the talus (OLT) is a relatively commonly cause of ankle pain and disability (Fig 1). Cartilage has very limited ability for repair or regeneration. A biologic solution
to the repair of significant cartilage defects is the “holy grail.” Some of the obstacles
to cartilage repair are the fact that it is highly vascular, hypocellular, and the
chondrocytes are “imprisoned” in a matrix (Fig 2).
Figure 1Magnetic resonance image of osteochondral lesions of the talus at the posteromedial
talar dome.
The fate of articular cartilage after transplantation of fresh and cryopreserved tissue-antigen-matched and mismatched osteochondral allografts in dogs.
The fate of cancellous and cortical bone after transplantation of fresh and frozen tissue-antigen-matched and mismatched osteochondral allografts in dogs.
The fate of articular cartilage after transplantation of fresh and cryopreserved tissue-antigen-matched and mismatched osteochondral allografts in dogs.
The fate of cancellous and cortical bone after transplantation of fresh and frozen tissue-antigen-matched and mismatched osteochondral allografts in dogs.
